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This glial metabolic deficiency deprives neurons of lactate-derived pyruvate for mitochondrial oxidative phosphorylation while concurrently impairing lactate-mediated epigenetic regulation of antioxidant genes (e.g., Nrf2), thereby synergistically exacerbating neuronal redox stress and bioenergetic collapse in mood-regulatory circuits [98,99]

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These trials, as well as studies aimed at determining the levels of GSH in the plasma of PAC-treated vs -untreated COVID-19 patients should be encouraged

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